C6 (complement component 6) is a critical component of the membrane attack complex (MAC), a multiprotein cascade activated by complement pathways 1. C6 associates with C5b at the outer leaflet of target cell membranes, reducing membrane bending energy and facilitating MAC assembly and insertion 12. This insertion creates pores in target cell membranes, leading to cell lysis and destruction of pathogens 34. The complement cascade, of which C6 is part, promotes pathogen phagocytosis and strengthens adaptive immunity 1. C6 exhibits genetic variation with common polymorphisms (C6A and C6B) maintained at similar frequencies across populations, suggesting balancing selection 5. C6 deficiency is an established disease association, impairing complement-mediated immune responses. Therapeutically, C6 inhibition shows promise for neurological diseases. A humanized monoclonal antibody against C6 (CP010) blocks the C6-C5/C5b interaction, preventing MAC formation and demonstrating efficacy in experimental autoimmune myasthenia gravis and autoimmune encephalomyelitis models 6. Animals deficient in C6 showed improved recovery after neuronal trauma, indicating that MAC inhibition may benefit neuronal regeneration 6. These findings position C6 as a potential therapeutic target for complement-mediated neuroinflammatory diseases.