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GeneE
6 sources retrieved Β· Most recent: April 2026 Β· Index updated 15 days ago
β“˜GeneE is for informational purposes only. It is not a substitute for professional medical advice, diagnosis, or treatment.
CCDC186
coiled-coil domain containing 186
Chromosome 10 Β· 10q25.3
NCBI Gene: 55088Ensembl: ENSG00000165813.20HGNC: HGNC:24349UniProt: A0A0C4DFU7
35PubMed Papers
20Diseases
0Drugs
1Pathogenic Variants
DATA QUALITY
βœ“ Experimental GO Evidenceβœ“ Swiss-Prot Reviewed
small GTPase bindingvesicle cytoskeletal traffickingtrans-Golgi networkGolgi apparatusFailure to thrivemicrocephalyhypothyroidismRelative macrocephaly
✦AI Summary

CCDC186 (coiled-coil domain containing 186) is a protein essential for dense-core vesicle (DCV) maturation in the trans-Golgi network of neurons and endocrine cells 1. The protein functions in small GTPase binding and vesicle cytoskeletal trafficking, facilitating the transport and proper maturation of DCVs, which are specialized organelles responsible for storing and releasing neurotransmitters and hormonal molecules 2. Loss-of-function variants in CCDC186 result in marked downregulation of gene expression and structural alterations affecting protein-protein interactions 2. Biallelic mutations in CCDC186 cause a novel autosomal recessive neurodevelopmental disorder characterized by seizures, developmental delay, epileptic encephalopathy, frontotemporal atrophy, hypomyelination, recurrent infections, failure to thrive, and endocrine disturbances including severe hypoglycemia and reduced cortisol or growth hormone levels 132. Clinical presentations typically manifest with neonatal onset 1. CCDC186 was identified as a candidate disease gene in a large diagnostic sequencing study and has now been confirmed through multiple case reports and functional studies demonstrating pathogenic effects of mutations 413. These findings establish CCDC186 as a critical mediator of neurodevelopmental and endocrine function.

Sources cited
1
CCDC186 is essential for DCV transport, causes neurodevelopmental phenotype with loss-of-function variants, downregulates gene expression, and affects protein-protein interactions
PMID: 40633195
2
CCDC186 involved in DCV maturation in trans-Golgi network; homozygous mutations cause seizures, atrophy, hypomyelination, infections, endocrine disturbances, and neonatal death
PMID: 37569695
3
Homozygous truncating CCDC186 variant causes epileptic encephalopathy, developmental delay, and failure to thrive
PMID: 33259146
4
CCDC186 identified as candidate disease gene in diagnostic sequencing study of 1000 families
PMID: 28600779
⚠Limited data available β€” This gene has 4 indexed publications. Summary and analysis may be incomplete.
Disease Associationsβ“˜20
Failure to thriveOpen Targets
0.34Weak
microcephalyOpen Targets
0.34Weak
Developmental stagnationOpen Targets
0.34Weak
EEG with spike-wave complexesOpen Targets
0.34Weak
Growth delayOpen Targets
0.34Weak
hypothyroidismOpen Targets
0.34Weak
pulmonary valve stenosisOpen Targets
0.34Weak
Relative macrocephalyOpen Targets
0.34Weak
SeizureOpen Targets
0.34Weak
Severe global developmental delayOpen Targets
0.34Weak
cardiovascular diseaseOpen Targets
0.21Weak
genetic disorderOpen Targets
0.19Weak
insomniaOpen Targets
0.17Weak
prostate carcinomaOpen Targets
0.08Suggestive
hypertensionOpen Targets
0.08Suggestive
breast cancerOpen Targets
0.06Suggestive
essential hypertensionOpen Targets
0.05Suggestive
cancerOpen Targets
0.03Suggestive
neoplasmOpen Targets
0.02Suggestive
Epileptic encephalopathyOpen Targets
0.01Suggestive
Pathogenic Variants1
NM_018017.4(CCDC186):c.767C>G (p.Ser256Ter)Pathogenic
10 conditions|not provided
β˜…β˜†β˜†β˜†2021β†’ Residue 256
View on ClinVar β†—
Related Genes
EIPR1Protein interaction72%
Tissue Expression6 tissues
Bone Marrow
100%
Heart
45%
Lung
28%
Brain
22%
Ovary
22%
Liver
17%
Gene Interaction Network
Click a node to explore
CCDC186EIPR1
PROTEIN STRUCTURE
Preparing viewer…
AlphaFoldAI-predicted Β· UniProt Q496Y1
View on AlphaFold β†—
Constraintβ“˜
LOEUFβ“˜
0.56Moderately Constrained
pLIβ“˜
0.02Tolerant
Observed/Expected LoF0.43 [0.34–0.56]
RankingsWhere CCDC186 stands among ~20K protein-coding genes
  • #10,928of 20,598
    Most Researched35
  • #5,211of 5,498
    Most Pathogenic Variants1
  • #3,638of 17,882
    Most Constrained (LOEUF)0.56 Β· top quartile
Genes detectedCCDC186
Sources retrieved6 papers
Response timeβ€”
πŸ“„ Sources
6β–Ό
1
The landscape of genetic diseases in Saudi Arabia based on the first 1000 diagnostic panels and exomes.
PMID: 28600779
Hum Genet Β· 2017
1.00
2
Biallelic novel CCDC186 loss-of-function variant disrupting the gene function causes neurodevelopmental phenotype and review of the literature.
PMID: 40633195
Brain Dev Β· 2025
0.83
3
Functional Evidence of
PMID: 37569695
Int J Mol Sci Β· 2023
0.67
4
Pitfalls in RET Fusion Detection Using Break-Apart FISH Probes in Papillary Thyroid Carcinoma.
PMID: 33382428
J Clin Endocrinol Metab Β· 2021
0.50
5
[Characteristics of RET gene rearrangement detected by fluorescence in situ hybridization in lung cancer].
PMID: 39762169
Zhonghua Bing Li Xue Za Zhi Β· 2025
0.33