HomeAboutRankingsData Sources
Β© 2026 GeneE
🧬
GeneE
10 sources retrieved Β· Most recent: April 2026 Β· Index updated 14 days ago
β“˜GeneE is for informational purposes only. It is not a substitute for professional medical advice, diagnosis, or treatment.
NOBOX
NOBOX oogenesis homeobox
Chromosome 7 Β· 7q35
NCBI Gene: 135935Ensembl: ENSG00000106410.15HGNC: HGNC:22448UniProt: A0A2R8Y8C8
28PubMed Papers
21Diseases
0Drugs
15Pathogenic Variants
FUNCTIONAL ROLE
Transcription Factor
CLINICAL
OMIM Disease Gene
DATA QUALITY
βœ“ Experimental GO Evidenceβœ“ Swiss-Prot Reviewed
regulation of transcription by RNA polymerase IIsequence-specific DNA bindingnucleusRNA polymerase II cis-regulatory region sequence-specific DNA bindingprimary ovarian insufficiencygenetic non-acquired premature ovarian failuregenetic disorderhereditary breast carcinoma
✦AI Summary

NOBOX (newborn ovary homeobox) is a gonad-specific transcription factor that plays a crucial role in early folliculogenesis and oogenesis 1. The gene encodes a homeobox-containing protein preferentially expressed in primordial and growing oocytes, with expression extending through the metaphase II stage 2. NOBOX binds preferentially to specific DNA sequences and functions as an RNA polymerase II-specific transcription factor regulating oocyte-specific gene expression 1. Biallelic and heterozygous loss-of-function variants in NOBOX are associated with primary ovarian insufficiency (POI), affecting approximately 1-2% of POI cases 3. Recent evidence indicates NOBOX variants account for about 25% of POI cases when considering ethnicity-dependent variant frequencies, though penetrance and expressivity vary significantly 4. Beyond POI, NOBOX variants have been identified in women experiencing oocyte, zygote, and embryo maturation arrest, suggesting roles in post-fertilization development 5. A corrected NOBOX gene model indicates that biallelic variants are most likely pathogenic, with only 14 of previously reported variants remaining causative for POI 6. NOBOX mutations represent a significant genetic contributor to female infertility, making genetic screening clinically relevant for patient management and counseling 7.

Sources cited
1
NOBOX is a homeobox-encoding gene preferentially expressed in primordial and growing oocytes with a role in early folliculogenesis
PMID: 11804785
2
Human NOBOX is oocyte-specific and expressed from primordial follicles through metaphase II oocytes
PMID: 16597639
3
NOBOX is implicated in ovarian development and meiosis pathways; associated with both syndromic and nonsyndromic POI
PMID: 34794894
4
NOBOX variants found in 25% of 810 POI patients; specific variants show ethnicity-dependent pathogenicity and variable penetrance
PMID: 37921973
5
Corrected NOBOX gene model indicates biallelic variants are most likely causative; only 14 of previously reported variants remain pathogenic
PMID: 40246288
6
NOBOX variants associated with oocyte, zygote, and embryo maturation arrest phenotype in addition to POI
PMID: 39871066
7
NOBOX variants identified in large cohort screening; genetic screening of POI patients is clinically worthwhile for molecular diagnosis
PMID: 36793102
8
NOBOX identified as a candidate gene with deleterious effect on POI, found in 1-2% of populations studied
PMID: 26243799
Disease Associationsβ“˜21
primary ovarian insufficiencyOpen Targets
0.78Strong
genetic non-acquired premature ovarian failureOpen Targets
0.49Moderate
genetic disorderOpen Targets
0.19Weak
Hereditary breast cancerOpen Targets
0.11Weak
hereditary breast carcinomaOpen Targets
0.11Weak
obesityOpen Targets
0.11Weak
osteoarthritisOpen Targets
0.09Suggestive
attention deficit hyperactivity disorderOpen Targets
0.07Suggestive
46,XX gonadal dysgenesisOpen Targets
0.06Suggestive
46,XX testicular disorder of sex developmentOpen Targets
0.06Suggestive
46,XY complete gonadal dysgenesisOpen Targets
0.06Suggestive
hereditary attention deficit-hyperactivity disorderOpen Targets
0.06Suggestive
attention deficit-hyperactivity disorder 8Open Targets
0.05Suggestive
intellectual disability, autosomal recessive 59Open Targets
0.05Suggestive
schizophrenia 15Open Targets
0.05Suggestive
partial androgen insensitivity syndromeOpen Targets
0.05Suggestive
azoospermiaOpen Targets
0.05Suggestive
spermatogenic failure 12Open Targets
0.05Suggestive
ring chromosome YOpen Targets
0.05Suggestive
Tourette syndromeOpen Targets
0.05Suggestive
Premature ovarian failure 5UniProt
Pathogenic Variants15
NM_001436401.1(NOBOX):c.652C>T (p.Arg218Ter)Pathogenic
Premature ovarian failure 5|not provided
β˜…β˜…β˜†β˜†2022β†’ Residue 218
NM_001080413.3(NOBOX):c.120T>A (p.Cys40Ter)Likely pathogenic
Premature ovarian failure 5
β˜…β˜†β˜†β˜†2024β†’ Residue 40
NM_001436401.1(NOBOX):c.1138del (p.Cys380fs)Likely pathogenic
NOBOX-related disorder
β˜…β˜†β˜†β˜†2023β†’ Residue 380
NM_001436401.1(NOBOX):c.958del (p.Val320fs)Pathogenic
Premature ovarian failure 5
β˜…β˜†β˜†β˜†2020β†’ Residue 320
NM_001436401.1(NOBOX):c.1089G>C (p.Lys363Asn)Likely pathogenic
Premature ovarian failure
β˜…β˜†β˜†β˜†2020β†’ Residue 363
NM_001436401.1(NOBOX):c.971del (p.Pro324fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2019β†’ Residue 324
NM_001436401.1(NOBOX):c.727C>T (p.Arg243Ter)Likely pathogenic
Premature ovarian failure 5
β˜…β˜†β˜†β˜†2019β†’ Residue 243
NM_001436401.1(NOBOX):c.696+23_696+24dupPathogenic
not provided
β˜…β˜†β˜†β˜†2019
NM_001436401.1(NOBOX):c.848C>T (p.Pro283Leu)Likely pathogenic
Genetic non-acquired premature ovarian failure
β˜†β˜†β˜†β˜†2019β†’ Residue 283
NM_001436401.1(NOBOX):c.947G>T (p.Gly316Val)Likely pathogenic
Genetic non-acquired premature ovarian failure
β˜†β˜†β˜†β˜†2019β†’ Residue 316
NM_001436401.1(NOBOX):c.780del (p.Gly261fs)Pathogenic
Genetic non-acquired premature ovarian failure
β˜†β˜†β˜†β˜†2019β†’ Residue 261
NM_001436401.1(NOBOX):c.1323_1326del (p.Phe442fs)Pathogenic
Genetic non-acquired premature ovarian failure
β˜†β˜†β˜†β˜†2019β†’ Residue 442
NM_001436401.1(NOBOX):c.1690dup (p.Ala564fs)Pathogenic
Genetic non-acquired premature ovarian failure
β˜†β˜†β˜†β˜†2019β†’ Residue 564
NM_001436401.1(NOBOX):c.697G>T (p.Val233Leu)Pathogenic
Premature ovarian failure 5
β˜†β˜†β˜†β˜†2011β†’ Residue 233
NM_001436401.1(NOBOX):c.696+74G>CPathogenic
Premature ovarian failure 5
β˜†β˜†β˜†β˜†2011
View on ClinVar β†—
Related Genes
ZP3Protein interaction99%SOHLH2Protein interaction99%FOXL2Protein interaction94%POF1BProtein interaction94%SOHLH1Protein interaction90%YBX2Protein interaction88%
Tissue Expression6 tissues
Bone Marrow
0%
Heart
0%
Ovary
0%
Liver
0%
Brain
0%
Lung
0%
Gene Interaction Network
Click a node to explore
NOBOXZP3SOHLH2FOXL2POF1BSOHLH1YBX2
PROTEIN STRUCTURE
Preparing viewer…
AlphaFoldAI-predicted Β· UniProt O60393
View on AlphaFold β†—
Constraintβ“˜
LOEUFβ“˜
1.06LoF Tolerant
pLIβ“˜
0.00Tolerant
Observed/Expected LoF0.81 [0.62–1.06]
RankingsWhere NOBOX stands among ~20K protein-coding genes
  • #12,401of 20,598
    Most Researched28
  • #2,482of 5,498
    Most Pathogenic Variants15
  • #10,669of 17,882
    Most Constrained (LOEUF)1.06
Genes detectedNOBOX
Sources retrieved10 papers
Response timeβ€”
πŸ“„ Sources
10β–Ό
1
Genetics of primary ovarian insufficiency: new developments and opportunities.
PMID: 26243799
Hum Reprod Update Β· 2015
1.00
2
Genetics of ovarian insufficiency and defects of folliculogenesis.
PMID: 34794894
Best Pract Res Clin Endocrinol Metab Β· 2022
0.90
3
Reclassifying NOBOX variants in primary ovarian insufficiency cases with a corrected gene model and a novel quantitative framework.
PMID: 40246288
Hum Reprod Β· 2025
0.80
4
Next-generation sequencing of 500 POI patients identified novel responsible monogenic and oligogenic variants.
PMID: 36793102
J Ovarian Res Β· 2023
0.70
5
Nobox is a homeobox-encoding gene preferentially expressed in primordial and growing oocytes.
PMID: 11804785
Mech Dev Β· 2002
0.60