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10 sources retrieved · Most recent: April 2026 · Index updated 15 days ago
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OCM
oncomodulin
Chromosome 7 · 7p22.1
NCBI Gene: 654231Ensembl: ENSG00000122543.11HGNC: HGNC:8105UniProt: P0CE72
17PubMed Papers
20Diseases
0Drugs
0Pathogenic Variants
DATA QUALITY
✓ Experimental GO Evidence✓ Swiss-Prot Reviewed
calcium ion bindingprotein bindingcytoplasmtype 1 diabetes mellitusplacenta praeviadeafnesshearing loss, autosomal recessive
✦AI Summary

Oncomodulin (OCM) is a calcium-binding protein with calmodulin-like activity involved in growth regulation and cell signaling. The protein binds two calcium ions and localizes to the cytoplasm where it exhibits protein-binding capabilities [UniProt]. OCM functions as a myeloid cell-derived growth factor that promotes axon regeneration following nerve injury in mice and rats 1. Mechanistically, OCM exerts its regenerative effects through binding to its high-affinity receptor ArmC10 (armadillo-repeat protein C10), which is expressed in sensory neurons and immune cells 1. ArmC10 deletion suppresses inflammation-induced axon regeneration in optic nerves and impairs the conditioning lesion effect for peripheral and spinal cord axon regeneration 1. Beyond nerve repair, OCM modulates immune function by increasing expression of immunomodulatory genes in human monocytes 1. In human-induced pluripotent stem cell-derived sensory neurons, OCM exposure alters gene expression and enhances neurite outgrowth 1. These findings suggest therapeutic potential for OCM-ArmC10 signaling in treating nerve injuries and modulating immune responses, though clinical translation requires further investigation.

Sources cited
1
Oncomodulin is a myeloid cell-derived growth factor enabling axon regeneration through binding to ArmC10 receptor; ArmC10 deletion suppresses nerve regeneration and OCM enhances neurite outgrowth in human sensory neurons
PMID: 37556559
⚠Limited data available — This gene has 1 indexed publication. Summary and analysis may be incomplete.
Disease Associationsⓘ20
type 1 diabetes mellitusOpen Targets
0.32Weak
placenta praeviaOpen Targets
0.23Weak
deafnessOpen Targets
0.12Weak
hearing loss, autosomal recessiveOpen Targets
0.11Weak
autosomal dominant nonsyndromic hearing lossOpen Targets
0.11Weak
neoplasmOpen Targets
0.10Suggestive
Non-syndromic genetic deafnessOpen Targets
0.08Suggestive
X-linked nonsyndromic hearing lossOpen Targets
0.07Suggestive
autosomal dominant nonsyndromic hearing loss 17Open Targets
0.07Suggestive
autosomal dominant nonsyndromic hearing loss 7Open Targets
0.07Suggestive
autosomal recessive nonsyndromic hearing loss 102Open Targets
0.07Suggestive
deafness, aminoglycoside-inducedOpen Targets
0.07Suggestive
Mitochondrial non-syndromic sensorineural deafness with susceptibility to aminoglycoside exposureOpen Targets
0.07Suggestive
autosomal recessive nonsyndromic hearing loss 86Open Targets
0.07Suggestive
hearing loss, autosomal dominant 88Open Targets
0.07Suggestive
hearing loss, autosomal dominant 89Open Targets
0.07Suggestive
auditory neuropathy, autosomal dominant 3Open Targets
0.07Suggestive
autosomal recessive nonsyndromic hearing loss 1BOpen Targets
0.07Suggestive
autosomal dominant nonsyndromic hearing loss 65Open Targets
0.07Suggestive
Meniere diseaseOpen Targets
0.07Suggestive
Pathogenic Variants
No pathogenic variants reported on ClinVar for this gene.
View on ClinVar ↗
Related Genes
SLC26A5Protein interaction90%PITPNM1Protein interaction83%GFI1Protein interaction73%
Tissue Expression6 tissues
Bone Marrow
100%
Brain
27%
Ovary
17%
Liver
8%
Lung
8%
Heart
0%
Gene Interaction Network
Click a node to explore
OCMSLC26A5PITPNM1GFI1
PROTEIN STRUCTURE
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PDB1TTX · NMR
View on RCSB ↗
Constraintⓘ
LOEUFⓘ
1.86LoF Tolerant
pLIⓘ
0.00Tolerant
Observed/Expected LoF1.35 [0.93–1.86]
RankingsWhere OCM stands among ~20K protein-coding genes
  • #15,080of 20,598
    Most Researched17
  • #16,937of 17,882
    Most Constrained (LOEUF)1.86
Genes detectedOCM
Sources retrieved10 papers
Response time—
📄 Sources
10▼
1
SCD1/FADS2 fatty acid desaturases equipoise lipid metabolic activity and redox-driven ferroptosis in ascites-derived ovarian cancer cells.
PMID: 35547771
Theranostics · 2022
1.00
2
In Vivo Imaging and Kinetic Modeling of Novel Glycogen Synthase Kinase-3 Radiotracers [
PMID: 37259346
Pharmaceuticals (Basel) · 2023
0.90
3
PMID: 30725915
0.80
4
Flavin-containing monooxygenase (FMO): Beyond xenobiotics.
PMID: 38713170
Bioessays · 2024
0.70
5
One-carbon metabolism and microbial pathogenicity.
PMID: 37224274
Mol Oral Microbiol · 2024
0.60