PKLR encodes pyruvate kinase L/R, a critical glycolytic enzyme catalyzing phosphoenolpyruvate conversion to pyruvate with ATP synthesis 1. This enzyme plays essential roles in both erythrocyte energy metabolism and hepatic glucose metabolism 2. Primary deficiency of PKLR causes congenital non-spherocytic hemolytic anemia due to impaired red blood cell glycolysis and energy depletion 13. Even never-transfused PK deficiency patients exhibit elevated comorbidities including osteoporosis, liver cirrhosis, and pulmonary hypertension 1. Regularly transfused patients face additional complications including iron overload and thrombosis 1. Beyond hemolysis, PKLR functions as a metabolic regulator in liver disease pathogenesis. PKLR is implicated in metabolic dysfunction-associated steatotic liver disease and hepatocellular carcinoma progression 2. In pancreatic cancer, HNF-1Ξ± transcriptionally upregulates PKLR to promote proliferation and apoptosis resistance 4. Genetically, PKLR variants exhibit pathogen-selection signatures. Certain haplotypes associated with malaria resistance paradoxically increase susceptibility to intracellular pathogens including Mycobacterium leprae and tuberculosis 5. Environmental stressors like dioxin exposure downregulate PKLR enzyme activity despite increased gene expression, affecting individuals in contaminated regions 6.
No tissue expression data available for this gene.