Unable to provide a gene function summary for CD101 based on the provided PubMed abstracts. The abstracts supplied focus exclusively on antifungal therapeutics (rezafungin, fosmanogepix, ibrexafungerp, olorofim), fungal infections (Candida, Aspergillus, Pneumocystis), and eosinophil biology—none of which contain information about CD101 molecule function. While one abstract 1 discusses CD101 as a surface marker distinguishing lung-resident eosinophil subsets (CD101lo versus CD101hi), this single reference provides insufficient data to establish CD101's primary function, mechanisms of T-cell inhibition, or clinical significance in disease contexts. To accurately summarize CD101's role as a T-cell proliferation inhibitor, its effects on IL-2 signaling, calcium mobilization, and potential involvement in leukemic disease markers, abstracts specifically investigating CD101 molecular mechanisms and disease associations would be required. The provided literature set does not support generating a comprehensive, evidence-based gene function summary.