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GeneE
10 sources retrieved Β· Most recent: April 2026 Β· Index updated 14 days ago
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MBTPS1
membrane bound transcription factor peptidase, site 1
Chromosome 16 Β· 16q23.3-q24.1
NCBI Gene: 8720Ensembl: ENSG00000140943.18HGNC: HGNC:15456UniProt: Q14703
86PubMed Papers
22Diseases
0Drugs
24Pathogenic Variants
FUNCTIONAL ROLE
Protease
CLINICAL
OMIM Disease Gene
DATA QUALITY
βœ“ Experimental GO Evidenceβœ“ Swiss-Prot Reviewed
serine-type endopeptidase activitymitochondrionprotein maturationGolgi membranespondyloepiphyseal dysplasia, Kondo-Fu typeneurodegenerative diseaseskeletal dysplasiasmoking initiation
✦AI Summary

MBTPS1 (membrane-bound transcription factor peptidase, site 1) is a serine protease located on chromosome 16 that catalyzes regulated intramembrane proteolysis of multiple substrates. Its primary function involves cleaving membrane-bound transcription factors after specific hydrophobic residue motifs 1. MBTPS1 catalyzes the initial proteolytic activation of sterol regulatory element-binding proteins (SREBPs) SREBF1/SREBP1 and SREBF2/SREBP2, which regulate cholesterol and lipid homeostasis 23. Additional substrates include activating transcription factor 6 (ATF6), CREB3L1, BDNF, and FAM20C, implicating MBTPS1 in ER stress response, bone mineralization, and lysosomal biogenesis 45. Mechanistically, MBTPS1 operates sequentially with site-2 protease (MBTPS2) to regulate cholesterol metabolism and endoplasmic reticulum stress responses 6. Notably, MBTPS1 also regulates antitumor immunity independently of its classical transcription factor cleavage function, competing with USP13 for STAT1 binding to modulate chemokine expression and CD8+ T cell infiltration 7. Pathogenic MBTPS1 variants cause multiple clinically distinct disorders including spondyloepiphyseal dysplasia (Kondo-Fu type), cataract-alopecia-oral mucosal disorder-psoriasis syndrome (CAOP), and Silver-Russell-like syndrome, with emerging phenotypes including white matter abnormalities and ectodermal dysplasia 16. MBTPS1 dysfunction disrupts lipid metabolism pathways critical for cancer cell proliferation, suggesting therapeutic potential for colorectal cancer 37.

Sources cited
1
MBTPS1 encodes site-1 protease that processes SREBPs, ATF6, and CREB3; variants cause multiple skeletal and skin disorders
PMID: 40877583
2
MBTPS1 catalyzes the first step in proteolytic activation of SREBP1, SREBP2, and ATF6
PMID: 12782636
3
MBTPS1 cleaves FAM20C propeptide, required for its secretion and kinase activity in osteoblast differentiation
PMID: 34349020
4
MBTPS1 mediates GNPTAB cleavage into subunits, participating in lysosomal biogenesis and M6P-dependent trafficking
PMID: 21719679
5
MBTPS1 regulates CRC proliferation primarily through SREBP activation; chemical inhibition decreases cell proliferation
PMID: 36300096
6
MBTPS1 inhibition enhances antitumor immunity by competing with USP13 for STAT1 binding, promoting chemokine expression and CD8+ T cell infiltration
PMID: 40307212
7
MBTPS1 variants cause autosomal recessive disorders with white matter loss, cataracts, and ectodermal dysplasia features
PMID: 38135440
Disease Associationsβ“˜22
spondyloepiphyseal dysplasia, Kondo-Fu typeOpen Targets
0.72Strong
neurodegenerative diseaseOpen Targets
0.50Moderate
skeletal dysplasiaOpen Targets
0.37Weak
smoking initiationOpen Targets
0.34Weak
mathematical abilityOpen Targets
0.22Weak
genetic disorderOpen Targets
0.19Weak
neoplasmOpen Targets
0.11Weak
Griscelli diseaseOpen Targets
0.09Suggestive
Griscelli disease type 3Open Targets
0.08Suggestive
Griscelli syndrome type 3Open Targets
0.08Suggestive
nail-patella syndromeOpen Targets
0.07Suggestive
diffuse large B-cell lymphomaOpen Targets
0.07Suggestive
infectionOpen Targets
0.07Suggestive
chronic obstructive pulmonary diseaseOpen Targets
0.07Suggestive
uncombable hair syndromeOpen Targets
0.07Suggestive
colorectal carcinomaOpen Targets
0.06Suggestive
Microcephaly - albinism - digital anomaliesOpen Targets
0.06Suggestive
microcephaly-albinism-digital anomalies syndromeOpen Targets
0.06Suggestive
glioblastoma multiformeOpen Targets
0.06Suggestive
gastrointestinal stromal tumorOpen Targets
0.06Suggestive
Cataract, alopecia, oral mucosal disorder, and psoriasis-like syndromeUniProt
Spondyloepiphyseal dysplasia, Kondo-Fu typeUniProt
Pathogenic Variants24
NM_003791.4(MBTPS1):c.2948G>A (p.Trp983Ter)Pathogenic
Spondyloepiphyseal dysplasia, kondo-fu type|See cases
β˜…β˜…β˜†β˜†2021β†’ Residue 983
NM_003791.4(MBTPS1):c.1018G>T (p.Gly340Ter)Pathogenic
not provided
β˜…β˜†β˜†β˜†2026β†’ Residue 340
NM_003791.4(MBTPS1):c.2432-1G>CLikely pathogenic
not provided
β˜…β˜†β˜†β˜†2026
NM_003791.4(MBTPS1):c.388C>T (p.Arg130Ter)Pathogenic
not provided
β˜…β˜†β˜†β˜†2025β†’ Residue 130
NM_003791.4(MBTPS1):c.846+1G>ALikely pathogenic
not provided
β˜…β˜†β˜†β˜†2025
NM_003791.4(MBTPS1):c.950del (p.Pro317fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2025β†’ Residue 317
NM_003791.4(MBTPS1):c.2413_2414del (p.Gln805fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2025β†’ Residue 805
NM_003791.4(MBTPS1):c.1391dup (p.Lys465fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2025β†’ Residue 465
NM_003791.4(MBTPS1):c.393del (p.Val132fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2025β†’ Residue 132
NM_003791.4(MBTPS1):c.1790dup (p.Asn597fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2024β†’ Residue 597
NM_003791.4(MBTPS1):c.721C>T (p.Arg241Ter)Pathogenic
not provided
β˜…β˜†β˜†β˜†2024β†’ Residue 241
NM_003791.4(MBTPS1):c.1781_1782del (p.Glu594fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2024β†’ Residue 594
NM_003791.4(MBTPS1):c.1601G>A (p.Trp534Ter)Pathogenic
not provided
β˜…β˜†β˜†β˜†2024β†’ Residue 534
NM_003791.4(MBTPS1):c.1824del (p.Ile609fs)Pathogenic
not provided
β˜…β˜†β˜†β˜†2024β†’ Residue 609
NM_003791.4(MBTPS1):c.1241G>C (p.Ser414Thr)Likely pathogenic
not provided
β˜…β˜†β˜†β˜†2024β†’ Residue 414
NM_003791.4(MBTPS1):c.2896C>T (p.Arg966Ter)Likely pathogenic
Spondyloepiphyseal dysplasia, kondo-fu type
β˜…β˜†β˜†β˜†2024β†’ Residue 966
NM_003791.4(MBTPS1):c.2567dup (p.Lys857fs)Likely pathogenic
not provided
β˜…β˜†β˜†β˜†2023β†’ Residue 857
NM_003791.4(MBTPS1):c.955G>T (p.Val319Phe)Likely pathogenic
Spondyloepiphyseal dysplasia, kondo-fu type
β˜…β˜†β˜†β˜†2020β†’ Residue 319
NM_003791.4(MBTPS1):c.1995C>G (p.Tyr665Ter)Pathogenic
Spondyloepiphyseal dysplasia, kondo-fu type
β˜…β˜†β˜†β˜†2020β†’ Residue 665
NM_003791.4(MBTPS1):c.1094A>G (p.Asp365Gly)Pathogenic
Spondyloepiphyseal dysplasia, kondo-fu type
β˜†β˜†β˜†β˜†2025β†’ Residue 365
View on ClinVar β†—
Related Genes
CREB3Protein interaction96%CREB3L1Protein interaction95%SREBF1Protein interaction95%SREBF2Protein interaction95%SCAPProtein interaction95%CREB3L3Protein interaction95%
Tissue Expression6 tissues
Heart
100%
Ovary
87%
Bone Marrow
64%
Lung
63%
Brain
53%
Liver
52%
Gene Interaction Network
Click a node to explore
MBTPS1CREB3CREB3L1SREBF1SREBF2SCAPCREB3L3
PROTEIN STRUCTURE
Preparing viewer…
PDB8UWC Β· 2.27 Γ… Β· EM
View on RCSB β†—
Constraintβ“˜
LOEUFβ“˜
0.85LoF Tolerant
pLIβ“˜
0.00Tolerant
Observed/Expected LoF0.70 [0.59–0.85]
RankingsWhere MBTPS1 stands among ~20K protein-coding genes
  • #5,548of 20,598
    Most Researched86
  • #2,016of 5,498
    Most Pathogenic Variants24
  • #7,389of 17,882
    Most Constrained (LOEUF)0.85
Genes detectedMBTPS1
Sources retrieved10 papers
Response timeβ€”
πŸ“„ Sources
10β–Ό
1
Inhibition of MBTPS1 enhances antitumor immunity and potentiates anti-PD-1 immunotherapy.
PMID: 40307212
Nat Commun Β· 2025
1.00
2
Discovery of a potent SCAP degrader that ameliorates HFD-induced obesity, hyperlipidemia and insulin resistance via an autophagy-independent lysosomal pathway.
PMID: 32432943
Autophagy Β· 2021
0.90
3
MBTPS1: a membrane-bound transcription factor protease implicated in the pathogenesis of several skin and skeletal disorders.
PMID: 40877583
Funct Integr Genomics Β· 2025
0.80
4
A case of MBTPS1-related disorder due to compound heterozygous variants in MBTPS1 gene: Genotype-phenotype expansion and the emergence of a novel syndrome.
PMID: 38135440
Am J Med Genet A Β· 2024
0.70
5
Cleavage of the soluble (pro)renin receptor (sATP6AP2) in the placenta.
PMID: 32920451
Placenta Β· 2020
0.60